In this BoardsCast episode, we continue our deep dive into Tobias Chapter 86 – Peritoneum & Retroperitoneum, expanding on how the peritoneum behaves as a biologic surface, not a “space,” and how that reality shapes the spread of infection, fluid, inflammation, and cancer.

Part B builds on the physiologic foundation from Part A and applies it to real clinical patterns, helping you understand why abdominal disease never behaves randomly — it follows rules.

You’ll learn:

• Why contamination spreads quickly while infection often stays localized
• How carcinoma “seeds” the abdomen along dependent surfaces
• Why fluid pockets form in predictable locations
• The difference between omental containment and omental failure
• Why retroperitoneal disease tracks along fascial planes instead of the abdomen
• How the peritoneum absorbs, filters, immunologically reacts, and sometimes overwhelms the body
• Board-relevant distinctions between free gas, free fluid, ascites, and septic pockets

If you’ve ever wondered why abdominal disease looks the way it does on CT or ultrasound, this episode gives you the framework.

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