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Risky Alzheimer's Genes and Unlocking Cells
On the 7th September 2020, Adam Smith hosted the Dementia Research Chatathon LIVE - to share information on the wide variety of research taking place across the world, and the raise money for Alzheimer's Research UK. Please continue to donate and help Adam meet his £5,000 target - details can be found at http://www.chatathon.uk.
In a way that everyone can understand, this video features Adam talking with:
Dr Hazel Hall-Roberts, Postdoctoral Researcher, University of Oxford (Now UK Dementia Research Institute, Cardiff University). I work on risk genes for Alzheimer's disease that are expressed highly in microglia, and I study them in a dish using human induced pluripotent stem cell models of microglia - @HazelLRoberts
Dr Mark Dallas, Associate Professor Neuroscience, University of Reading. My work looks at non neuronal cells and their potential to combat Alzheimer's. Cellular work looking a ion channels on glial cells and changes in their function long before clinical presentation. In addition, we are looking at the ability of carbon monoxide to suppress amyloid toxicity and be a neuroprotectant - @drmarkdallas
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By Adam SmithOn the 7th September 2020, Adam Smith hosted the Dementia Research Chatathon LIVE - to share information on the wide variety of research taking place across the world, and the raise money for Alzheimer's Research UK. Please continue to donate and help Adam meet his £5,000 target - details can be found at http://www.chatathon.uk.
In a way that everyone can understand, this video features Adam talking with:
Dr Hazel Hall-Roberts, Postdoctoral Researcher, University of Oxford (Now UK Dementia Research Institute, Cardiff University). I work on risk genes for Alzheimer's disease that are expressed highly in microglia, and I study them in a dish using human induced pluripotent stem cell models of microglia - @HazelLRoberts
Dr Mark Dallas, Associate Professor Neuroscience, University of Reading. My work looks at non neuronal cells and their potential to combat Alzheimer's. Cellular work looking a ion channels on glial cells and changes in their function long before clinical presentation. In addition, we are looking at the ability of carbon monoxide to suppress amyloid toxicity and be a neuroprotectant - @drmarkdallas