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Aronia berry supplementation supports gut, arterial healthKing’s College London, October 17 2022.

The issue of Clinical Nutrition reported findings from a randomized, double-blind, placebo-controlled trial that uncovered a benefit for supplementation with aronia berry among men and women with prehypertension.

“Aronia melanocarpa, or black chokeberry, has gained increased attention for its high content of (poly)phenols, and potential protection against chronic diseases such as cardiovascular disease and diabetes,” authors Melanie Le Sayec of King’s College London and colleagues remarked.

The trial included 102 participants between the ages of 40 and 70 years who received aronia berry extract capsules that contained 106 milligrams total polyphenols or identical placebo capsules for 12 weeks. Blood pressure, arterial function, gut microbiome composition and other factors were assessed before and after the treatment period.

At the end of the study, augmentation index and pulse wave velocity, which measure arterial stiffness, significantly improved among participants who received aronia in comparison with the placebo group. The aronia-supplemented group also had a significant increase in microbiome species richness and abundance of species that produce butyrate, a compound that has a favorable effect in gut health.

The researchers observed associations between plasma and urinary aronia-derived polyphenol metabolites, decreased arterial stiffness and various gut flora species. “To our knowledge, this is the first time these species are shown to correlate with beneficial effects on arterial function,” they announced.

“The present findings suggest that daily consumption of aronia berry extract led to improvements in arterial function in healthy middle-aged people, with a concomitant and related increase in potentially health-promoting bacterial taxa,” the authors concluded.

Zinc enhances albumin’s protective role against Parkinson’s diseaseUniversity of Science and Technology, October 17, 2022

Revealing zinc’s interaction with a critical transport protein underscores the need to study biological pathways under physiologically relevant conditions.

Heavy metals in the body have long been thought to induce the aggregation of disease-linked proteins, but a KAUST study shows this is not always the case.

It turns out that zinc ions tune the ability of human serum albumin (HSA), an abundant transport protein in the body, to better prevent α-synuclein from aggregating, a process directly linked to Parkinson’s disease.

In unrealistically massive quantities, zinc tends to accelerate the aggregation of α-synuclein, a neuronal protein implicated in Parkinson’s disease. This is what other scientists had shown in the past. But, under the types of biologically plausible experimental conditions considered by Al-Harthi, the metal actually interacts with HSA to cause the opposite effect.

Using a state-of-the-art imaging technique known as proton-less nuclear magnetic resonance spectroscopy, Al-Harthi and colleagues showed that zinc binding alters the chaperone function of HSA, a multifunctional molecule that plays a role in blocking α-synuclein accretion. In particular, zinc ions biased HSA toward stronger interactions with the aggregation-prone fragments of α-synuclein, a change that blunts fibril formation and slows down the toxic process of protein deposition that can lead to neurodegeneration.

High exposure to glyphosate in pregnancy could cause lower birth weights in babiesIndiana University School of Medicine, October 17, 2022

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