Videos:

A.I. Is Making it Easier to Kill (You). Here’s How. | NYT (10:00)

Disturbing simulation shows power, terror of killer robots (7:46)

Fear Psychosis and the Cult of Safety – Why are People so Afraid? (13:25)

 

Peanuts Linked to Lower Breast Cancer Risk

Washington University in St. Louis & Harvard University, October 22, 2022

Girls ages 9 to 15 who regularly ate peanut butter or nuts were 39 percent less likely to develop benign breast disease by age 30, according to a new study.

Benign breast disease, although noncancerous, increases risk of breast cancer later in life.

“These findings suggest that peanut butter could help reduce the risk of breast cancer in women,” says senior author Graham Colditz, professor of surgery at Washington University School of Medicine and associate director for cancer prevention and control at Siteman Cancer Center at Barnes-Jewish Hospital.

Published in the journal Breast Cancer Research and Treatment, the findings are based on the health histories of 9,039 US girls enrolled in the Growing Up Today Study .

When the study participants were 18 to 30 years old, they reported whether they had been diagnosed with benign breast disease that had been confirmed by breast biopsy.

Participants who ate peanut butter or nuts two times each week were 39 percent less likely to have developed benign breast disease than those who never ate them.

The study’s findings suggest that beans, lentils, soybeans, and corn also may help prevent benign breast disease, but consumption of these foods was much lower in these girls so the evidence was weaker.

Past studies have linked peanut butter, nut, and vegetable fat consumption to a lower risk for benign breast disease. However, participants in those studies were asked to recall their high school dietary intakes years later.

Perinatal Brain DHA Concentration Has a Lasting Impact on CognitionNational Institute on Alcohol Abuse and Alcoholism October 20, 2022

A new study on Proteomics is now available. According to news out of Bethesda, Maryland, research stated, “Premature infants are deprived of prenatal accumulation of brain docosahexaenoic acid [DHA (22: 6n-3)], an omega-3 fatty acid [omega-3 FA (n-3 FA)] important for proper development of cognitive function. The resulting brain DHA deficit can be reversed by omega-3 FA supplementation.”

“The objective was to test whether there is a critical period for providing omega-3 FA to correct cognitive deficits caused by developmental omega-3 FA deprivation in mice. Twelve timed-pregnant mice were fed an omega-3 FA-deficient diet containing 0.04% a-linolenic acid, and their offspring were fed the same deficient diet (Def group) or changed to an omega-3 FA-adequate diet containing 3.1% ALA at 3 wk, 2 mo, or 4 mo of age. In parallel, 3 E14 pregnant mice were fed the adequate diet and their offspring were fed the same diet (Adeq group) throughout the experiment. Brain FA composition, learning and memory, and hippocampal synaptic protein expression were evaluated at 6 mo by gas chromatography, the Morris water maze test, and western blot analysis, respectively.

Maternal dietary omega-3 FA deprivation decreased DHA by > 50% in the brain of their offspring at 3 wk of age. The Def group showed significantly worse learning and memory at 6 mo than those groups fed the adequate diet. These pups also had decreased hippocampal expression of postsynaptic density protein 95 (43% of Adeq group), Homer protein homolog 1 (21% of Adeq group), and synaptosome-associated protein of 25 kDa (64% of Adeq group).

Changing mice to the adequate diet at 3 wk, 2 mo, or 4 mo of age restored brain DHA to the age-matched adequate concentration. However, deficits in hippocampal synaptic protein expression and spatial learning and memory were normalized only when the diet was changed at 3 wk.”

The research concluded: “Developmental deprivation of brain DHA by dietary omega-3 FA depletion in mice may have a lasting impact on cognitive function