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Mice missing the autism-linked SHANK2 and SHANK3 genes in their retrosplenial cortex have trouble distinguishing between novel and familiar mice.
Compared with their unaffected siblings and unrelated controls, children with autism harbor more copy number variants in genes that govern the circadian cycle or are associated with insomnia.
Common variants in five regions of the genome may determine whether someone has one condition versus the other.
The transplanted cells integrate into living animals’ neural circuitry and influence behavior.
Researchers can maximize the impact of their results by engaging policymakers more effectively.
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