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Curious why coconut oil can raise LDL for some people yet still show up in diets that improve metabolic health, cognition, and body composition? We get clear on what coconut oil actually is—a family of saturated fats with a rare abundance of medium-chain triglycerides—and why MCTs create ketones that the brain and mitochondria can use even without fasting. Instead of stopping at cholesterol, we look at the fuller picture: insulin resistance, inflammation, triglycerides, and real-world outcomes that drive risk more than a single lab value.

We dig into the differences between MCT oil, extra-virgin coconut oil, and refined coconut oil, and when each makes sense. We compare biomarkers to hard endpoints, explain why some populations with high coconut intake have low cardiovascular disease, and explore how diet context flips the script: in a high-sugar diet, lipids can look worse; in a lower-carb, whole-food pattern, markers of metabolic syndrome often improve even if LDL nudges up. Along the way, we share insights on cooking stability, oxidative stress, and the intriguing idea that latitude and season may influence how fats signal thermogenesis and photoprotection.

You’ll leave with practical takeaways you can use today: how to choose between MCTs and whole coconut oil, how to integrate them into a low-sugar, nutrient-dense diet, and which labs to track beyond LDL, like ApoB, triglyceride-to-HDL ratio, hs-CRP, and fasting insulin. If you’ve wondered whether coconut oil is a villain or a misunderstood ally, this conversation offers a grounded, outcome-focused way to decide what fits your body and your climate.

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