Hu Said: Cardiology Board Review Series

In this episode, we review right side congenital abnormalities, including Ebstein’s anomaly, double-chambered right ventricle (DCRV), tricuspid hypoplasia, and pulmonic hypoplasia. Which condition is characterized by "atrialization" of the right ventricle? How does the apical displacement of the septal leaflet of the tricuspid valve in Ebstein anomaly compare with what occurs in atrioventricular septal defect? What is the significance of an accessory pathway in patients with Ebstein’s anomaly? What are the indications for surgical intervention in Ebstein’s anomaly? How can tricuspid valve dysplasia be distinguished from Ebstein’s anomaly? What is the role of right ventricle-to-pulmonary artery conduits, and when should they be replaced? Tune in for answers to these questions.

Difficulty Level: Intermediate

You are listening to Hu Said: Cardiology Board Review Series (2024-2025 Season)

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In this episode, we review Tetralogy of Fallot (TOF), including its pre-repair issues, surgical options, and post-repair complications. What are the four cardinal abnormalities seen in TOF, and how do they impact right-to-left shunting and systemic oxygenation? What are the differences between the Blalock-Taussig shunt, the Waterston or Potts shunts, and complete TOF repair? What surgical strategies are used today? When are temporizing procedures acceptable over complete repair? What are the long-term complications after TOF repair, including pulmonary regurgitation (PR), arrhythmias, and sudden cardiac death, and how are these managed? When is it acceptable to perform pulmonary valve replacement in post-TOF repair patients who have moderate or severe PR that is asymptomatic? Tune in for answers to these questions.

Difficulty Level: Advanced

You are listening to Hu Said: Cardiology Board Review Series (2024-2025 Season)

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In this episode, we review coarctation of the aorta (CoA), including its epidemiology, presentation, imaging, and management. Which type of VSD is commonly seen in patients with CoA? What proportion of CoA patients have bicuspid aortic valve, and what proportion of bicuspid aortic valve patients have CoA? What key physical exam findings suggest CoA, and how does it present in young patients versus adults? What pattern should you see on PW Doppler of the descending aorta and CW Doppler of the coarctation? What is considered a significant peak-to-peak gradient across a coarctation? What are the criteria for surgical or transcatheter intervention? What are the long-term complications after repair? When should you suspect re-coarctation after CoA repair? Tune in for answers to these questions.

Difficulty Level: Intermediate

You are listening to Hu Said: Cardiology Board Review Series (2024-2025 Season)

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In this episode, we review the physiology, imaging, and management of patent ductus arteriosus (PDA). What is the classic murmur of PDA, and how does it change with shunt reversal? Why do patients with PDA get left heart enlargement instead of right heart enlargement? How does the differential cyanosis seen in PDA with Eisenmenger syndrome present? How do you calculate pulmonary artery systolic pressure using PDA velocity? How do you distinguish the Doppler CW profile of PDA from pulmonary valve regurgitation? What are the indications for PDA closure, and why are Qp ratios not used as they are for ASD and VSD? Tune in for answers to these questions.

Difficulty Level: Intermediate

You are listening to Hu Said: Cardiology Board Review Series (2024-2025 Season)

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In this episode, we explore anomalous pulmonary venous return (APVR), including its types, associated conditions, and management. What is the difference between total anomalous pulmonary venous return (TAPVR) and partial anomalous pulmonary venous return (PAPVR)? How do supracardiac, cardiac, infracardiac, and mixed types of TAPVR differ? Which ASD type is most commonly associated with PAPVR? What are the physiologic consequences of right heart overload caused by APVR, and what are the criteria for surgical intervention? How is Scimitar syndrome diagnosed and managed, and when should surgery be considered for this rare condition? Lastly, we will touch upon persistent superior vena cava syndrome. Tune in for answers to these questions.

Difficulty Level: Advanced

You are listening to Hu Said: Cardiology Board Review Series (2024-2025 Season)

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In this episode, we explore atrioventricular septal defects (AVSD), covering its types, physiology, and when to intervene. How do complete, partial, transitional, and intermediate AV septal defects differ in structure and physiology? What are the anatomic and functional consequences of a primum ASD with cleft mitral valve, and how does this affect the mitral valve's position relative to the tricuspid valve? What challenges do patients with AVSD face post-surgery, including risks of mitral regurgitation, left ventricular outflow tract obstruction, and arrhythmias? When is surgery recommended for AVSD, and what are the key considerations for long-term follow-up? Tune in for answers to these questions.

Difficulty Level: Advanced

You are listening to Hu Said: Cardiology Board Review Series (2024-2025 Season)

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In this episode, we review ventricular septal defects (VSDs), including their types, physiology, and management. What are the differences between inlet, outlet, membranous, and trabecular VSDs? How do these defects vary in terms of location, association with other cardiac conditions, and suitability for percutaneous closure? Which VSD types tend to have aortic cusp prolapse, leading to aortic regurgitation? If you see a color jet from a VSD in parasternal short axis view, what is the significance of the color jet being adjacent to the tricuspid valve versus being adjacent to the pulmonary valve? What are the physiologic consequences of left-to-right shunting in VSDs, and how does this lead to left heart dilation, pulmonary overcirculation, and potential progression to Eisenmenger syndrome? What are the criteria for VSD closure? Which is the only VSD type that is amenable to percutaneous closure? Tune in for answers to these questions.

Difficulty Level: Advanced

You are listening to Hu Said: Cardiology Board Review Series (2024-2025 Season)

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In this episode, we review patent foramen ovale (PFO), including its types, physiology, presentation, and when to intervene. How does a PFO differ from an atrial septal defect (ASD), and why does PFO not cause the right heart enlargement and pulmonary hypertension seen in ASD? What is platypnea-orthodeoxia syndrome? What can cause platypnea-orthodeoxia syndrome other than PFO? What features on brain MRI suggest that the etiology of an ischemic stroke is cardioembolic? What features on echocardiography distinguish between PFOs and ASDs? How can PFOs contribute to cryptogenic stroke, and what high-risk features indicate the need for closure? Tune in for answers to these questions.

Difficulty Level: Intermediate

You are listening to Hu Said: Cardiology Board Review Series (2024-2025 Season)

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In this episode, we review atrial septal defects (ASDs), including their types, imaging, and management. What is the difference between secundum defect, primum defect, sinus venosus defect, and coronary sinus defect? Which is the only ASD defect out of the four that can be percutaneously closed (in addition to surgically closed)? Which ASD type is accompanied by anomalous pulmonary venous return? What are the physiologic consequences of left-to-right shunting in ASDs, and how can these lead to pulmonary hypertension and RV failure? What is Holt-Oram syndrome? What are the criteria for ASD closure? Tune in for answers to these questions.

Difficulty Level: Advanced

You are listening to Hu Said: Cardiology Board Review Series (2024-2025 Season)

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More MedEd resources available at ⁠www.rueyhu.com⁠

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In this episode, we review the epidemiology of congenital heart diseases and develop a framework for categorizing them by cyanotic and non-cyanotic disorders, and go over physiologic concepts across congenital conditions. What are the 5 'T' disorders that are characterized by cyanosis? What are the non-T disorders that can develop cyanosis? What is the tipping point for Eisenmenger syndrome to occur? What are the non-cardiac manifestations of Eisenmenger syndrome? Is phlebotomy appropriate in Eisenmenger syndrome? What is the role of bosentan and sildenafil/tadalafil in Eisenmenger syndrome? What Qp/Qs ratio indicates a large left-to-right shunt that requires repair? What Qp/Qs ratio indicates a a small left-to-right shunt? What Qp/Qs ratio indicates net right-to-left shunting? What is the Flamm formula for calculating mixed venous saturation proximal to a transatrial shunt? What should you suspect if pulmonary oxygen saturation exceeds 80%, or is 8% higher than the superior vena cava's oxygen saturation? What is the difference between physiologic shunting and anatomic shunting? What conditions comprise ACHD physiologic stages A, B, C, and D? Tune in for answers to these questions.

Difficulty Level: Intermediate

You are listening to Hu Said: Cardiology Board Review Series (2024-2025 Season)

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More MedEd resources available at ⁠www.rueyhu.com⁠

Updates at Twitter/X: ⁠@Ruey_Hu⁠